InvivoGen's product citations
InvivoGen’s products are frequently cited in peer-reviewed publications for a wide variety of applications and uses. Browse our product citations below.
Please note: By clicking on the product name, you can access via the ‘Citations’ tab all references of the product (click ‘load more’).
Citations search
You can search by ‘Product Name’ or ‘Year’.
| Product | Citation | Year | Journal | Authors | Page |
|---|---|---|---|---|---|
| QUANTI-Luc™: Luciferase Detection Reagent | Trypanosoma brucei Invariant Surface Glycoprotein 75 Is an Immunoglobulin Fc ... | 2024 | J Immunol. | Mikkelsen J.H. et al. | DOI: 10.4049/jimmunol.2300862 |
| CpG-free HSV1-tk | Effect of Different Karyophilic Peptides on Physical Characteristics and In V... | 2024 | Mol Biotechnol. | Aranda-Barradas M.E. et al. | DOI: 10.1007/s12033-024-01087-9 |
| CpG-free HSV1-tk::Sh | Effect of Different Karyophilic Peptides on Physical Characteristics and In V... | 2024 | Mol Biotechnol. | Aranda-Barradas M.E. et al. | DOI: 10.1007/s12033-024-01087-9 |
| RAW-Blue™ Cells | Self-adjuvanted L-arginine-modified dextran-based nanogels for sustained loca... | 2024 | Biomater Sci. | Chung J.T. et al. | DOI: 10.1039/d3bm01150j |
| Normocin® - Antimicrobial Reagent | The O-glycan is essential for the induction of protective antibodies against ... | 2024 | Infect Immun. | Choi M. et al. | DOI: 10.1128/iai.00427-23 |
| Human TLR5 Reporter HEK293 Cells (NF-κB) | The O-glycan is essential for the induction of protective antibodies against ... | 2024 | Infect Immun. | Choi M. et al. | DOI: 10.1128/iai.00427-23 |
| QUANTI-Blue™ | The O-glycan is essential for the induction of protective antibodies against ... | 2024 | Infect Immun. | Choi M. et al. | DOI: 10.1128/iai.00427-23 |
| Human TLR4 Reporter HEK293 Cells (NF-κB) | The O-glycan is essential for the induction of protective antibodies against ... | 2024 | Infect Immun. | Choi M. et al. | DOI: 10.1128/iai.00427-23 |
| Anti-β-Gal-IgA2 isotype control | Bacillus subtilis Induces Human Beta Defensin-2 Through its Lipoproteins in H... | 2024 | Probiotics Antimicrob Proteins | So Y.J. et al. | DOI: 10.1007/s12602-024-10224-4 |
| Anti-hTLR2 Detection and Neutralizing mAb | Bacillus subtilis Induces Human Beta Defensin-2 Through its Lipoproteins in H... | 2024 | Probiotics Antimicrob Proteins | So Y.J. et al. | DOI: 10.1007/s12602-024-10224-4 |
| G3-YSD | Chronic endoplasmic reticulum stress in myotonic dystrophy type 2 promotes au... | 2024 | Nat Commun. | Rösing S. et al. | DOI: 10.1038/s41467-024-45535-1 |
| THP1-Dual™ Cells | Chronic endoplasmic reticulum stress in myotonic dystrophy type 2 promotes au... | 2024 | Nat Commun. | Rösing S. et al. | DOI: 10.1038/s41467-024-45535-1 |
| IFN-α/β Reporter HEK 293 Cells | Chronic endoplasmic reticulum stress in myotonic dystrophy type 2 promotes au... | 2024 | Nat Commun. | Rösing S. et al. | DOI: 10.1038/s41467-024-45535-1 |
| QUANTI-Luc™: Luciferase Detection Reagent | Chronic endoplasmic reticulum stress in myotonic dystrophy type 2 promotes au... | 2024 | Nat Commun. | Rösing S. et al. | DOI: 10.1038/s41467-024-45535-1 |
| Poly(I:C) HMW | Chronic endoplasmic reticulum stress in myotonic dystrophy type 2 promotes au... | 2024 | Nat Commun. | Rösing S. et al. | DOI: 10.1038/s41467-024-45535-1 |
| 3'3'-cGAMP | DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response. | 2024 | Cell Death Differ. | Jiao H. et al. | DOI: 10.1038/s41418-024-01269-7 |
| c-di-GMP | DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response. | 2024 | Cell Death Differ. | Jiao H. et al. | DOI: 10.1038/s41418-024-01269-7 |
| 2'3'-cGAMP | DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response. | 2024 | Cell Death Differ. | Jiao H. et al. | DOI: 10.1038/s41418-024-01269-7 |
| 5'ppp-dsRNA | DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response. | 2024 | Cell Death Differ. | Jiao H. et al. | DOI: 10.1038/s41418-024-01269-7 |
| Zeocin® | Naphthoquinone derivatives as potential immunomodulators: prospective for COV... | 2024 | RSC Adv. | Moraes V.T. et al. | DOI: 10.1039/d3ra08173g |